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dc.contributor.authorRizk, Nina Camilleen_US
dc.date.accessioned2017-09-21T17:20:44Z
dc.date.available2017-09-21T17:20:44Z
dc.date.issued2017
dc.identifier.urihttps://hdl.handle.net/2144/23847
dc.description.abstractMental illness is currently diagnosed using subjective observational criteria as outlined in the 5th Edition of the Diagnostic and Statistical Manual (DSM-V), yet many have argued for the medicalization of the diagnosis of mental illness by incorporating biomedical and neuroanatomical criteria. The following literature review explores the neural circuit responsible for regulating emotion, as well as the structural and chemical alterations to this circuit that have been shown to correlate with aggressive and/or violent behaviors characteristic of certain types of mental illness. The neural circuit regulating emotion is comprised of the prefrontal cortex, the subcortical limbic system, the dopaminergic pathway, the serotonergic pathway, catecholaminergic neurons, and GABAergic neurons. Alterations to these structures or chemicals have been associated with major depressive disorder, suicidal ideations, substance use disorders, schizophrenia, and personality disorders. Medicalization of mental illness has the potential to serve two purposes – first, to standardize diagnosis and treatment of mental illness, and second, to decrease the stigma often associated with mental illness – and to improve outcomes for those patients living with mental illness.en_US
dc.language.isoen_US
dc.rightsAttribution 4.0 Internationalen_US
dc.rights.urihttp://creativecommons.org/licenses/by/4.0/
dc.subjectMental healthen_US
dc.subjectAggressionen_US
dc.subjectViolenceen_US
dc.subjectNeural circuiten_US
dc.titleAggressive and violent behavior - the result of malfunction in the neural circuit regulating emotionen_US
dc.typeThesis/Dissertationen_US
dc.date.updated2017-07-13T19:26:16Z
etd.degree.nameMaster of Scienceen_US
etd.degree.levelmastersen_US
etd.degree.disciplineMedical Sciencesen_US
etd.degree.grantorBoston Universityen_US


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Attribution 4.0 International
Except where otherwise noted, this item's license is described as Attribution 4.0 International